By American Academy of Ophthalmology, Ramana S. Moorthy MD
Starts off with an outline of immune-mediated eye illness, summarizing uncomplicated immunologic strategies, ocular immune responses and unique subject matters in ocular immunology. Discusses the scientific method of uveitis and reports noninfectious (autoimmune) and infectious types of uveitis, with an elevated part on viral uveitis and new fabric on infectious and noninfectious scleritis.
Enhanced detection of infectious brokers by way of immunologic and genetic equipment and biologic therapeutics are certain. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular points of AIDS.
Upon of completion of part nine, readers might be capable to:
Identify common and particular pathophysiologic procedures that have an effect on the constitution and serve as of the uvea, lens, intraocular cavities, retina, and different tissues in acute and persistent intraocular inflammation
Differentiate and establish infectious and noninfectious uveitic entities
Choose applicable exam innovations and suitable ancillary experiences according to no matter if an infectious or noninfectious reason is suspected
Read or Download 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis PDF
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Extra resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis
Major histocompatibility complex (MHC) class I molecules (ie, HLA-A, -B, and -C) serve as the antigen-presenting platform for CDS T lymphocytes (Fig 2-2). CDS T lymphocytes include natural killer T cells and regulatory T cells. Class I molecules are present on almost all nucleated cells. In general, class I APCs are best for processing peptide antigens that have been synthesized by the host cell itself, including most tumor peptides or viral peptides after host cell invasion. MHC class II molecules (ie, HLA-DR, -DP, and-DQ) serve as the antigen-presenting platform for CD4, or helper, T lymphocytes (Fig 2-3).
Some of the free toxin or hapten is also carried by lymph into the node. In the lymph node, the processing phase begins. The urushiol-stimulated LCs interact with T lymphocytes, seeking over the next 3-5 days the rare T lymphocyte that has the correct specific antigen receptor. Once located, this naive T lymphocyte becomes primed. It is induced to undergo cell division, to acquire new functions such as cytokine secretion, and to up-regulate certain surface molecules and receptors of the plasma membrane.
Which can inhibit or alter the effector function of other T lymphocytes. Regulatory T lymphocytes are potentially important as they may provide a means for induction of tolerance to specific antigens. 42 • lntraocular Inflammation and Uveitis Effector Phase (eg, · th e e fc1ec t or p h ase, the adaptive immune response D unng . . the elimination . of offend. · c · t' ) · physically carried out. Antigen-speofrc effectors exist m 2 maJor mg 1ore1gn an 1gen 1s subsets: • T lymphocytes • B lymphocytes plus their antibodies In general, effector lymphocytes require 2 exposures to antigen.